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Monday, July 27, 2020 | History

2 edition of Effects of temporary tourniquet ischemia on striated muscle fibers and motor end-plates. found in the catalog.

Effects of temporary tourniquet ischemia on striated muscle fibers and motor end-plates.

Lars-Olof DahlbaМ€ck

Effects of temporary tourniquet ischemia on striated muscle fibers and motor end-plates.

Morphological and histochemical studies in the rabbit and electromyographical studies in man.

by Lars-Olof DahlbaМ€ck

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  • 5 Currently reading

Published by Almqvist & Wiksell (distr.) in Stockholm .
Written in

    Subjects:
  • Ischemia.,
  • Striated muscle.,
  • Neuromuscular transmission.,
  • Electromyography.,
  • Histocytochemistry.,
  • Ischemia.,
  • Muscles.,
  • Neuromuscular junction.

  • Edition Notes

    Bibliography: p. 88-91.

    SeriesScandinavian journal of plastic and reconstructive surgery. Supplementum,, 7
    Classifications
    LC ClassificationsRB144 .D35
    The Physical Object
    Pagination91 p.
    Number of Pages91
    ID Numbers
    Open LibraryOL4781482M
    LC Control Number75502065

    striated muscle. fibres and motor. end. Open muscle biopsies were taken from tibialis anterior muscle immediately after tourniquet release 24, 36 and 48 hours after tourniquet release. phosphokinase release as a measure of tourniquet effect on skeletal muscle. Arch Surg ;(1): Dahlback L 0. Effects of temporary tourniquet ische- mia on striated muscle fibers and motor end plates. Morphological and histochemical studies in the rabbit and electromyographical studies in man. Scand J PIast.

    Injection of a mitochondria-targeted S-nitrosation enhancing agent, MitoSNO, into ischemic muscle prior to reperfusion reduced mitochondrial oxidative stress in the motor nerve and NMJ, attenuated. The adverse effects of excessive tourniquet pressure on underlying muscle were studied using a cat model. A direct effect of the magnitude of tourniquet pressure on the degree of muscle disability.

    The effects of tourniquet release on systemic blood pressure and metabolic parameters were studied in 11 adult patients undergoing total knee replacement under general anesthesia. Mean arterial pressure (MAP) decreased rapidly after the release of the tourniquet, becoming significant at 3 min and remaining significantly depressed up to 15 min.   Effects of temporary tourniquet ischemia on striated muscle fibers and motor end-plates. Morphological and histochemical studies in the rabbit and electromyographical studies in man. Dahlbäck LO. Scand J Plast Reconstr Surg Suppl, , 01 Jan Cited by 6 .


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Effects of temporary tourniquet ischemia on striated muscle fibers and motor end-plates by Lars-Olof DahlbaМ€ck Download PDF EPUB FB2

Scand J Plast Reconstr Surg Suppl. ; Effects of temporary tourniquet ischemia on striated muscle fibers and motor end-plates. Morphological and histochemical studies in the rabbit and electromyographical studies in by:   REFERENCES I.

Dahlback LO: Effects of temporary tourniquet ischemia on striated muscle fibers and motor end plates. Scand J Plastic Reconstr Surg Suppl 1: 2. Harman JW: A histologic study of skeletal muscle in acute ischemia. Am J Patholby: Effects of temporary tourniquet ischemia on striated muscle fibers and motor end-plates.

Morphological and histochemical studies in the rabbit and electromyographical studies in man. Tourniquet application is a widely accepted adjuvant technique in extremity surgery. The purpose of this prospective, randomized trial was to evaluate the effect of cuff width on skeletal muscle.

Ultrastructural examination of the intrinsic hand muscles was performed on young healthy patients undergoing reconstructive operations following tendo Cited by: 7. Temporary ischaemia of the hind limb of the rat was produced using a tourniquet with controlled pressure.

Changes in the muscle seen after Gomori trichrome staining and after histochemical reactions for NADHdiaphorase, ATPases and phosphorylase were correlated with the duration of the ischaemia and the time of recovery. Histopathological changes were seen first after 2 h of ischaemia and.

Tourniquet use in total knee arthroplasty (TKA) surgery is applied to minimize blood loss thereby creating better overview of the surgical field. This induces ischemia in the skeletal muscle resulting in reperfusion injury. Our aim was to investigate the in vivo metabolic changes in the skeletal muscle during TKA surgery using microdialysis (MD).

Seventy patients were randomly allocated to. Introduction. Extremity trauma constitutes the majority of war wounds, both historically, and during the current wars in Iraq and Afghanistan, and is a significant problem in civilian of these wounds include muscle trauma, often involving acute ischemia reperfusion injury (IRI) due to vascular injury, emergency tourniquet (TK) application, and/or acute extremity compartment.

Using a validated model of tourniquet-induced-acute murine hindlimb ischemia-reperfusion (Crawford et al., ), this study investigated the prophylactic effects of co-enzyme Q 10 and superoxide dismutase mimetic (tempol) on the ischemia-reperfusion-induced skeletal muscle infarct size and mitochondrial dysfunction to clarify if superoxide.

In mouse models of familial amyotrophic lateral sclerosis (fALS) motor neurons are especially vulnerable to oxidative stresses in vitro. To determine whether this increased vulnerability also extends to motor nerve terminals in vivo, we assayed the effect of tourniquet-induced ischemia/reperfusion (I/R) injury on motor terminals innervating fast and slow hindlimb muscles in male G93A-SOD1 mice.

To determine whether this increased vulnerability also extends to motor nerve terminals in vivo, we assayed the effect of tourniquet-induced ischemia/reperfusion (I/R) injury on motor terminals.

The jitter (the variability in time interval between the action potentials from 2 muscle fibers belonging to the same motor unit) has been used to study the neuro-muscular transmission in individual motor end-plates. Ischemia increased the jitter and caused more and more frequent blocking of the neuro- muscular transmission until total block.

Effects of temporary tourniquet ischemia on striated muscle fibers and motor end-plates. Morphological and histochemical studies in the rabbit and electromyographical studies in man.

Dahlbäck LO. Scand J Plast Reconstr Surg Suppl,01 Jan Cited by: 6 articles | PMID: Influence of fiber-type composition on recovery from tourniquet-induced skeletal muscle ischemia–reperfusion injury.

Thomas J. Walters, a John F. Kragh, a David G. Baer a. a United States Army Institute of Surgical Research, Rawley E. Chambers Ave. Dahlbäck L () Effects of temporary tourniquet ischaemia on striated muscle fibers and motor end-plates.

Scand Plast Reconstr Surg Suppl Google Scholar Sarkar M. Degeneration of a part of the presynaptic nerve endings of motor end-plates with or without simultaneous degeneration of the postsynaptic muscle fibre was seen after ischaemia lasting 3 h or longer.

Motor end-plates were first seen on the 45th day in the muscle subjected to 6 h ischaemia and were most likely regenerated ones. Effects of temporary tourniquet ischemia on striated muscle fibers and motor end-plates. Morphological and histochemical studies in the rabbit and electromyographical studies in man.

Scand J Plast Reconstr Surg Suppl. ; –   Recovery of skeletal muscle function is of prime importance in revascularization of acutely ischemic extremities. In order to study the effect of ischemia and reperfusion on peripheral nerve and skeletal muscle function, temporary unilateral hindlimb ischemia (1 or 3 hr) was induced in 12 rabbits by temporary arterial occlusion of common iliac and femoral arteries and collateral ligation.

The structure and distribution of the capillaries of the rat anterior tibial muscle were analyzed by electronmicroscopy and microangiography 2 h and 1, 4, 18, 45 and 90 days after temporary.

Author(s): Dahlbäck,Lars-Olof Title(s): Effects of temporary tourniquet ischemia on striated muscle fibers and motor end-plates; morphological and histochemical studies in the rabbit and electromyographical studies in man. Country of Publication: Sweden Publisher: Umeå. In mouse models of familial amyotrophic lateral sclerosis (fALS), motor neurons are especially vulnerable to oxidative stresses in vitro.

To determine whether this increased vulnerability also extends to motor nerve terminals in vivo, we assayed the effect of tourniquet-induced ischemia/reperfusion (I/R) injury on motor terminals innervating fast and slow hindlimb muscles in .Local effects are the result of tissue ischaemia distal to the inflated tourniquet and a combination of ischaemia and compression of the tissues beneath it.

Muscle Following inflation of the tourniquet, there is a progressive decrease in P o 2 and an increase in P co 2 within muscle cells.The purpose of varicosities in motor nerve fibers in muscle physiology is A.

to link the thin filaments to the inside of the sarcolemma in smooth muscle B. to reabsorb the decomposition products of acetylcholine after acetylcholinesterase breaks it down C. to enable each cardiac myocyte to directly stimulate its neighbors.